Glycogen synthase kinase (GSK) 3beta hyperactivity impairs glomerular podocyte insulin signaling via IRS1 modulation in diabetic kidney disease
DOI:
https://doi.org/10.46570/utjms-2025-1352Keywords:
GSK3beta hyperactivity, hyperinsulinemia, diabetic kidney diseaseDownloads
Published
2025-06-30
How to Cite
Chen, M., Ge, Y., Dworkin, L., & Gong, R. (2025). Glycogen synthase kinase (GSK) 3beta hyperactivity impairs glomerular podocyte insulin signaling via IRS1 modulation in diabetic kidney disease. Translation: The University of Toledo Journal of Medical Sciences, 13(S3). https://doi.org/10.46570/utjms-2025-1352
Issue
Section
Dr. Lance D. Dworkin Department of Medicine Research Symposium
License
Copyright (c) 2025 Mengxuan Chen, Yan Ge, Lance Dworkin, Rujun Gong (Author)
This work is licensed under a Creative Commons Attribution 4.0 International License.
Authors who publish with this journal agree to the following terms:
- Authors retain copyright and grant the journal right of first publication with the work simultaneously licensed under a Creative Commons Attribution License (CC BY 4.0) that allows others to share the work with an acknowledgement of the work's authorship and initial publication in this journal.
- Authors are able to enter into separate, additional contractual arrangements for the non-exclusive distribution of the journal's published version of the work (e.g., post it to an institutional repository or publish it in a book), with an acknowledgement of its initial publication in this journal.
- Authors are permitted and encouraged to post their work online (e.g., in institutional repositories or on their website) prior to and during the submission process, as it can lead to productive exchanges, as well as earlier and greater citation of published work (See The Effect of Open Access).
