Glycogen synthase kinase (GSK) 3beta hyperactivity impairs glomerular podocyte insulin signaling via IRS1 modulation in diabetic kidney disease

Authors

  • Mengxuan Chen The University of Toledo Department of Medicine
  • Yan Ge Division of Nephrology, University of Toledo College of Medicine
  • Lance Dworkin Division of Nephrology, University of Toledo College of Medicine https://orcid.org/0000-0002-2471-5989
  • Rujun Gong Division of Nephrology, University of Toledo College of Medicine

DOI:

https://doi.org/10.46570/utjms-2025-1352

Keywords:

GSK3beta hyperactivity, hyperinsulinemia, diabetic kidney disease

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Published

2025-06-30

How to Cite

1.
Chen M, Ge Y, Dworkin L, Gong R. Glycogen synthase kinase (GSK) 3beta hyperactivity impairs glomerular podocyte insulin signaling via IRS1 modulation in diabetic kidney disease. Translation [Internet]. 2025 Jun. 30 [cited 2025 Aug. 12];13(S3). Available from: http://openjournals.utoledo.edu/index.php/translation/article/view/1352

Issue

Vol. 13 No. S3 (2025): The 2024 Dr. Lance D. Dworkin Department of Medicine Research Symposium

Section

Dr. Lance D. Dworkin Department of Medicine Research Symposium

License

Copyright (c) 2025 Mengxuan Chen, Yan Ge, Lance Dworkin, Rujun Gong (Author)

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